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Addressing questions on GDD

1. My opinion, based on research to date, is that GDD has a genetic basis. A number of sufferers have taken exception and have been alarmed by this, with their impression that I am saying that they are responsible for their own disease - that they are guilty. This is absolutely not the case. My opinion is that perhaps essentially all diseases have some form of genetic basis, which varies from relatively minor to very strong. BRCA1 and BRCA2 and breast cancer (and other cancers); colon cancer; polyposis syndromes and colon cancer; to name just a few. Because you have a genetic predisposition to the disease, neither makes it your fault, nor does it mean you should be ignored and not treated. W

Association Versus Causation GDD

Association versus causation is always a huge issue in medicine (and other disciplines). Quite often when something is found to be present at the same time as a disease, many scientists/physicians start cautiously by claiming association before being prepared to assert causation. This certainly was the case with Gd and NSF, when for years from 2006 onwards a number of experts struggled with the association vs causation. Association means that things have been found together, which is very different from something directly resulting in the disease. Gd does cause NSF. This certainly is where many experts are at with GDD, wanting to consider the relationship an association rather causation. Th

The Complement System: Let me help with some proteins

I have written a number of blogs on the immune system. I have simplified the discussion focusing primarily on the timing of activity of members of the immune system, as this explains differences between the 3 major forms of gadolinium toxicity: acute hypersensitivty reaction, GDD, and NSF. Essentially the immune system is also categorized into the innate immune system and the adaptive system. The most basic difference is that the innate system are designed to kill invaders and don't show much variation. The key members are neutrophils, which act early, and the principle combatants with infection (also result in the formation of pus), and macrophages which handle invaders in a more subacute f

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