All readers of my blogs know that a central focus has been on Gd. But also that we intend to deal with all heavy metals, especially in the nonprofit GadTTRAC. So to date: Gd- most of blogs. Pb (lead) - probably same reservoirs and most of identical symptoms as GDD. Also DTPA + Frame best method to treat. Thallium and Cesium - major source presently kale. Starting point don't eat kale. Treatment likely a combination of Prussian Blue and probably DTPA as well. Now mercury. The

These are the 5 Horsemen of the GDD Flare Apocalypse: initial deposition, stationary presence, removal, redistribution, re-equilibration. Below they are listed and strategies to manage them. Initial Deposition. Management: health care workers and patients must recognize GDD when it first arises after a GBCA injection. The principle treatment for GDD, as for most toxic exposures, is to never get another GBCA agent again. If GDD is recognized when it first occurs, often subtle

Essentially almost by definition patients with GDD have normal/near normal renal function. Yet a number of individuals do have renal abnormalities, but not sufficient to cause renal failure, but I think possibly sufficient to cause subtle abnormalities in kidney handling of Gd. One such renal abnormality which I have observed in a few patients is medullary sponge kidney. Amongst other things, this abnormality has a high incidence of small calcified renal stones. Perhaps there

Integrative physicians are the ones who recognized the disease GDD early on - earlier than me - so I will always hold them in esteem for that. Maybe now actually I am one of their number. One issue that I am struck by is the lack of knowledge by essentially the vast majority of treating practitioners in the fields of heavy metals and chelation: the stability constant of the chelator and the heavy metal is of paramount importance and must be known in order to use a chelator T